A novel scientific review published in the Journal of Translational Medicine has revealed the importance of mitochondrial function in women's reproductive health.1
Covering the role of mitochondrial dysfunction in the decline of oocyte quality and ovarian reserve, the review assesses how mitochondrial health can impact reproductive wellness and general health.
The review also covers the potential of targeting the mitochondria with therapeutics as a way to address women's reproductive health.
Amid these findings, Australian clinician Dr Leah Hechtman will give a talk at the Complementary Medicines Australia (CMA) Innovation Day in Sydney on May 14, where she will discuss the role of mitochondrial function in supporting women's health across all life and fertility stages.
Dr Hechtman will also discuss the role of ubiquinol, an antioxidant sometimes used in nutraceutical products, in supporting mitochondrial function.
"Women's health across the lifespan is closely linked to mitochondrial vitality," noted Dr Hechtman.
"However, modern lifestyle factors such as stress, exposure to environmental toxins, poor nutrition, long-term fatigue and hormonal fluctuations can compromise mitochondrial efficiency,"
"This has the capacity to impact overall fertility, vitality and general wellbeing," she stated.
However, by supporitng mitochondrial health with Ubiquinol, consumers can assist the body in self-regulation and responding to hormonal changes.
"Ubiquinol is naturally produced by the body, and plays a central role in mitochondrial energy pathways by facilitating electron transport for ATP production," comments Dr Hechtman.
As ubiquinol levels start to decline after the age of 20, the body can be exposed to higher levels of oxidative stress, which impacts energy production and overall mitochondrial health.
"For women, this is significant, as efficient cellular energy production underpins key physiological processes such as hormonal production and regulation — both essential components of overall reproductive health”, Dr Hechtman concluded.
Reference
1 https://translational-medicine.biomedcentral.com/articles/10.1186/s12967-025-06223-w#Sec9